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Management of Malunited Fractures: Comprehensive Surgical Principles and Techniques

01 May 2026 20 min read 50 Views
Management of Malunited Fractures: Comprehensive Surgical Principles and Techniques

Key Takeaway

Malunion occurs when a fracture heals in a non-anatomic position, leading to altered biomechanics, functional impairment, and premature arthrosis. Successful management requires meticulous preoperative planning, precise osteotomy execution, and robust fixation. This guide details the principles of malunion reconstruction across major anatomical regions, emphasizing the role of conventional internal fixation and advanced circular frame techniques for complex deformities involving bone loss or prior infection.

Comprehensive Introduction and Patho-Epidemiology

A malunion is strictly defined as a fracture that has healed in a non-anatomic position, resulting in a complex, multi-planar deformity that may be characterized by angular, rotational, translational, or length-discrepancy components. While the human skeleton exhibits some capacity to tolerate minor degrees of malalignment—contingent upon the specific anatomical region, the patient's age, and their physiological demands—significant malunions fundamentally alter joint reaction forces. This biomechanical disruption impairs physiological kinematics, drastically increases focal cartilage contact pressures, and predictably initiates the cascade of premature post-traumatic arthrosis (PTOA). The surgical management of malunited fractures remains one of the most intellectually and technically demanding endeavors in the field of orthopedic surgery, demanding a profound understanding of three-dimensional spatial geometry.

The pathophysiology of malunion is intrinsically linked to the initial fracture pattern, the chosen method of primary stabilization, and the biological environment of the healing bone. Epidemiologically, malunions are most frequently encountered in the distal radius, the tibial diaphysis, and the clavicle. Distal radius malunions often result from the collapse of osteoporotic bone following non-operative management or the failure to recognize comminution that necessitates rigid internal fixation. In the lower extremity, tibial malunions frequently follow the non-operative management of spiral or oblique fractures, or they occur secondary to dynamically locked intramedullary nails that fail to control rotational or angular forces during the consolidation phase. The incidence of malunion is also significantly elevated in patients with severe polytrauma, where the necessity for rapid, life-saving damage-control orthopedics may temporarily supersede the goal of perfect anatomic reduction.

Understanding the patho-epidemiology requires an appreciation of the temporal evolution of the deformity. A malunion is not merely a static structural anomaly; it is a dynamic biomechanical lesion. For example, a varus malunion of the tibial diaphysis exceeding 5 degrees shifts the mechanical axis deviation (MAD) medially. This shift exponentially increases the load transmitted through the medial compartment of the knee, overwhelming the viscoelastic dampening properties of the articular cartilage and menisci. Over time, this leads to subchondral sclerosis, cartilage fibrillation, and eventual full-thickness chondral loss. Furthermore, chronic deformities induce secondary soft-tissue contractures; muscles, tendons, and neurovascular structures adapt to the shortened or angulated skeletal architecture, making delayed acute surgical correction highly perilous and technically fraught.

The primary objective of malunion reconstructive surgery extends far beyond cosmetic restoration. It is a joint-preserving, function-restoring intervention designed to re-establish the mechanical axis, restore joint congruity, and balance the soft-tissue envelope. By normalizing the joint reaction forces and restoring the physiological lever arms of the surrounding musculature, the orthopedic surgeon can arrest the progression of degenerative joint disease, alleviate chronic pain, and dramatically improve the patient's functional capacity. This requires meticulous preoperative planning, a mastery of both internal and external fixation techniques, and an unwavering commitment to the principles of deformity correction.

Detailed Surgical Anatomy and Biomechanics

A rigorous comprehension of surgical anatomy and biomechanics is the absolute foundation of successful deformity correction. The surgeon must evaluate the limb not just as a singular bone, but as an integrated mechanical linkage system. In the lower extremity, this begins with the definition of the mechanical axis, which is a straight line drawn from the center of the femoral head to the center of the ankle joint (the center of the talar dome). In a normal, well-aligned lower extremity, this mechanical axis line should pass precisely through the center of the knee joint, or slightly medial to the tibial spines. Any deviation of this line, termed the Mechanical Axis Deviation (MAD), quantifies the magnitude of the mechanical overload on the respective joint compartment.

To precisely localize the apex of the deformity, the surgeon must utilize the Center of Rotation of Angulation (CORA) methodology, initially popularized by Dror Paley. The CORA is the intersection point of the proximal and distal anatomical (or mechanical) axis lines of the deformed bone. Understanding the relationship between the osteotomy site, the CORA, and the chosen axis of correction is paramount. According to Paley’s rules: if the osteotomy and the hinge of correction are both located at the CORA (Rule 1), pure angular correction is achieved without translation. If the osteotomy is performed away from the CORA, but the hinge remains at the CORA (Rule 2), the correction will result in a planned, collinear translation that restores the overall axis. Failure to respect these geometric rules (Rule 3) results in iatrogenic translation, creating a secondary deformity that can further compromise limb biomechanics and soft-tissue coverage.

In the upper extremity, the biomechanical paradigm shifts from weight-bearing axial alignment to complex rotational kinematics and prehension. The forearm functions as a highly sophisticated joint system where the radius rotates around the relatively fixed ulna. The anatomical bow of the radial diaphysis is critical for generating the clearance necessary for full pronation and supination. A diaphyseal malunion that flattens this radial bow, or alters the complex tensioning of the interosseous membrane (particularly its central band), will result in a profound mechanical block to rotation. Similarly, in the distal radius, the normal volar tilt (average 11 degrees) and radial inclination (average 22 degrees) dictate the kinematics of the radiocarpal and distal radioulnar joints (DRUJ). A dorsal malunion (Colles-type) shifts the carpal load dorsally, increases forces on the ulna (ulnar impaction syndrome), and distorts the sigmoid notch, leading to DRUJ incongruity, subluxation, and severe functional impairment.

The soft-tissue envelope is an equally critical anatomical consideration that dictates the surgical approach and the limits of acute correction. Chronic malunions are invariably accompanied by adaptive shortening of the neurovascular bundles, musculotendinous units, and the cutaneous envelope. Acute correction of a long-standing angular or length deformity places immense traction on these structures. For instance, acute correction of a severe valgus knee deformity can stretch the common peroneal nerve, leading to devastating iatrogenic palsy. Similarly, extensive periosteal stripping during the surgical approach can devascularize the bone ends, transforming a malunion into a recalcitrant nonunion. Therefore, the surgeon must deeply understand the angiosomes of the limb, utilizing internervous and intermuscular planes, and frequently employing gradual correction via circular external fixation when the soft-tissue limits preclude acute intra-operative realignment.

Exhaustive Indications and Contraindications

The decision to proceed with the surgical correction of a malunited fracture is highly individualized, requiring a meticulous risk-benefit analysis that weighs the severity of the deformity against the patient's physiological age, functional demands, and medical comorbidities. The primary indication for surgical intervention is a symptomatic deformity that causes intractable pain, mechanical block to motion, or significant gait dysfunction. In the lower extremity, absolute indications include intra-articular step-offs greater than 2 millimeters in weight-bearing joints (e.g., tibial plateau, distal tibia), as these predictably lead to rapid joint destruction. Extra-articular deformities are indicated for correction if they cause a mechanical axis deviation that significantly overloads a single compartment, typically defined as angular deformities exceeding 5 to 10 degrees in the coronal or sagittal planes, or rotational deformities exceeding 15 degrees that result in severe out-toeing or in-toeing, leading to compensatory hip and spine pathology.

In the upper extremity, indications are driven by pain, loss of grip strength, and restriction of activities of daily living. For distal radius malunions, surgical correction is strongly indicated for young, active patients presenting with >15 degrees of dorsal tilt, >3 mm of radial shortening (resulting in positive ulnar variance and ulnar impaction), or symptomatic DRUJ incongruity. Forearm diaphyseal malunions that restrict pronosupination to less than 50 degrees in either direction are also prime candidates for corrective osteotomy. Furthermore, limb length discrepancies exceeding 2.0 to 2.5 centimeters in the lower extremity generally warrant surgical intervention, either through lengthening of the malunited segment, acute shortening of the contralateral limb, or shoe lifts, depending on the patient's preference and overall height.

Contraindications to malunion surgery are equally critical to recognize to prevent catastrophic outcomes. Absolute contraindications include active, uncontrolled deep infection (unless the surgery is specifically designed as an eradication procedure utilizing debridement and circular external fixation), severe peripheral vascular disease that precludes adequate healing of the osteotomy and soft tissues, and profound neuropathic arthropathy (Charcot joint) where reconstructive efforts are doomed to fail due to lack of protective sensation. Relative contraindications encompass advanced age with low functional demands, severe osteoporosis that compromises hardware purchase, active smoking (which exponentially increases the risk of nonunion), and a lack of patient psychological readiness or compliance, particularly when complex postoperative rehabilitation or external fixation frames are planned.

Category Specific Indications Specific Contraindications
Lower Extremity (Extra-articular) Coronal/Sagittal angulation > 5-10°; Rotational deformity > 15°; Limb length discrepancy > 2.5 cm; Symptomatic mechanical axis deviation. Advanced, end-stage post-traumatic osteoarthritis (better suited for arthroplasty); Severe peripheral vascular disease.
Lower Extremity (Intra-articular) Articular step-off or gap > 2 mm; Joint instability secondary to bony deformity; Impending localized cartilage wear. Active deep osteomyelitis (relative - dictates fixation type); Charcot neuroarthropathy.
Upper Extremity (Distal Radius) Dorsal tilt > 15°; Radial shortening > 3 mm; Symptomatic DRUJ incongruity; Loss of > 50% normal ROM. Asymptomatic elderly patient with low functional demands; Severe CRPS (Complex Regional Pain Syndrome).
Upper Extremity (Diaphyseal) Loss of radial bow preventing pronosupination; Symptomatic shortening of the clavicle > 2 cm with shoulder dyskinesia. Poor soft tissue envelope precluding safe surgical approach; Non-compliant patient.
Systemic / Patient Factors High functional demands; Intractable pain; Impending adjacent joint degeneration. Uncontrolled psychiatric illness; Active heavy smoking (relative); Severe medical comorbidities (ASA IV).

Pre-Operative Planning, Templating, and Patient Positioning

Pre-operative planning is the most critical phase of malunion surgery; the operation is largely won or lost before the first incision is made. The clinical evaluation must be exhaustive. The surgeon must meticulously observe the patient's gait, looking for antalgic patterns, varus or valgus thrusts, and compensatory mechanisms such as pelvic obliquity or circumduction. The rotational profile of the lower extremities must be quantified using the thigh-foot angle and hip internal/external rotation arcs to differentiate between femoral and tibial torsional deformities. The soft-tissue envelope must be scrutinized; previous surgical incisions, areas of split-thickness skin grafting, and adherent, avascular scars dictate the choice of the surgical approach. A history of prior infection necessitates a comprehensive workup, including inflammatory markers (ESR, CRP) and potentially a preoperative aspiration or bone biopsy, as this drastically alters the fixation strategy from internal hardware to external circular frames.

Radiographic evaluation demands standardized, high-quality imaging. For lower extremity deformities, full-length, weight-bearing, standing anteroposterior radiographs (from hip to ankle) are mandatory. These films allow the surgeon to perform the Malalignment Test, calculating the MAD, the joint orientation angles (e.g., mechanical lateral distal femoral angle [mLDFA], medial proximal tibial angle [MPTA]), and precisely locating the CORA. Computed Tomography (CT) is indispensable, particularly for intra-articular malunions (e.g., tibial plateau, distal radius, calcaneus) and complex multi-planar diaphyseal deformities. CT scans with 3D reconstructions allow the surgeon to visualize the spatial architecture of the deformity, assess articular step-offs, and measure rotational malalignment by superimposing proximal and distal reference axes (e.g., femoral neck axis vs. posterior condylar axis).

Modern reconstructive surgery heavily relies on digital templating and advanced computer-assisted planning. Utilizing specialized software, the surgeon can virtually perform the osteotomy, manipulate the fragments, and template the exact size and trajectory of the internal fixation devices. In highly complex cases, 3D printing is utilized to create life-size biomodels of the malunited bone. This allows the surgeon to physically practice the osteotomy and pre-contour plates prior to sterilization, saving invaluable tourniquet time. Furthermore, Patient-Specific Instrumentation (PSI)—custom-designed 3D-printed cutting jigs—can be manufactured based on the CT data to guide the exact angle and plane of the osteotomy intra-operatively, virtually eliminating the guesswork associated with multi-planar corrections.

Patient positioning and operating room setup must be meticulously choreographed to facilitate unimpeded fluoroscopic imaging and optimal surgical access. The patient is typically positioned on a completely radiolucent Jackson table or a flat OSI table. For lower extremity procedures, a sterile bump is often placed under the ipsilateral hip to correct external rotation, and a radiolucent triangle is kept on the sterile field to position the knee at various degrees of flexion. The C-arm fluoroscope must be positioned such that it can easily arc from AP to lateral without compromising the sterile field. Tourniquets are applied but used judiciously, often inflated only during the critical exposure and osteotomy phases to minimize ischemic time, and deflated prior to closure to ensure meticulous hemostasis, thereby reducing the risk of postoperative hematoma and subsequent infection.

Step-by-Step Surgical Approach and Fixation Technique

Lower Extremity Malunions

Femoral malunions frequently present with a complex triad of shortening, angulation, and malrotation. For diaphyseal femoral malunions, the surgical approach typically involves a lateral incision to access the apex of the deformity. The correction requires a meticulously planned diaphyseal osteotomy. If the deformity is purely rotational or involves a simple angular correction without significant translation, a transverse or short oblique osteotomy is performed. Intramedullary nailing (either antegrade or retrograde, depending on the location of the CORA) is the gold standard for fixation. The use of blocking screws (Poller screws) placed in the concavity of the deformity is often mandatory to narrow the medullary canal and direct the nail, preventing the deformity from reproducing as the nail is passed. For subtrochanteric malunions resulting in coxa vara, a lateral approach is utilized to perform a closing wedge or dome osteotomy, restoring the neck-shaft angle. Rigid fixation is achieved using a dynamic condylar screw (DCS) or a proximal femoral locking plate, ensuring robust purchase in the femoral head and neck.

Tibial diaphyseal malunions are notoriously unforgiving due to the subcutaneous nature of the medial tibial face and the parallel orientation of the knee and ankle hinge joints. Once the CORA is identified, the osteotomy is executed. For midshaft deformities, a percutaneous or minimally invasive transverse osteotomy can be performed, followed by reamed intramedullary nailing. However, for proximal or distal metaphyseal malunions where nail control is tenuous, plate osteosynthesis is preferred. An opening wedge osteotomy is often utilized to simultaneously correct angulation and restore length, with the resulting defect filled with structural allograft or autologous iliac crest bone graft. The construct is stabilized with a pre-contoured locking compression plate (LCP). In cases involving severe multi-planar deformities, poor soft tissue, or a history of osteomyelitis, the Ilizarov apparatus or Taylor Spatial Frame (TSF) is the definitive treatment. A percutaneous Gigli saw osteotomy or multiple drill-hole corticotomy is performed to preserve the endosteal and periosteal blood supply, followed by gradual computer-assisted distraction osteogenesis.

Foot and ankle malunions require highly specialized salvage techniques. Talar neck malunions typically heal in varus, leading to lateral border overload and a rigid, supinated foot. The surgical approach involves a medial utility incision, performing a medial opening wedge osteotomy of the talar neck, inserting a structural bone graft, and stabilizing with headless compression screws. Calcaneal malunions, characterized by loss of height, increased width, and subfibular impingement, are approached via an extensile lateral incision. The surgeon performs a lateral wall exostectomy to decompress the peroneal tendons, followed by a subtalar arthrodesis. If the talocalcaneal angle is severely flattened, a distraction bone block arthrodesis is performed, utilizing a tricortical iliac crest graft to restore calcaneal height and correct the varus deformity of the tuberosity.

Upper Extremity Malunions

Distal radius malunions demand precise restoration of volar tilt, radial inclination, and ulnar variance to salvage wrist kinematics. For the classic extra-articular malunion with dorsal angulation (Colles-type), a volar (modified Henry) approach is utilized. The flexor carpi radialis (FCR) sheath is opened, the FCR tendon is retracted ulnarly, and the pronator quadratus is elevated from its radial border. A critical step is the release of the brachioradialis insertion from the radial styloid; failure to perform this release will tether the distal fragment, making restoration of radial length impossible. A transverse osteotomy is performed parallel to the articular surface at the site of the previous fracture using an oscillating saw. The distal fragment is mobilized volarly and ulnarly. The resulting volar cortical gap is meticulously filled with a structural autograft (iliac crest) or a synthetic bone substitute. Rigid fixation is achieved with a volar locking plate, which buttresses the graft and allows for immediate postoperative digital range of motion.

Forearm diaphyseal malunions severely restrict pronation and supination due to the disruption of the complex rotational axis. The surgical strategy hinges on restoring the precise anatomical bow of the radius. Osteotomies are performed at the apex of the deformity through standard volar (Henry) or dorsal (Thompson) approaches for the radius, and a direct subcutaneous approach for the ulna. Because diaphyseal cortical bone heals slowly, an opening wedge or step-cut osteotomy is preferred to maximize surface area contact. Rigid fixation utilizing 3.5mm dynamic compression plates (DCP) or locking compression plates (LCP) is absolute. Due to the high risk of nonunion at these diaphyseal osteotomy sites, the application of cancellous autograft or demineralized bone matrix (DBM) around the osteotomy is routinely performed. In cases of cross-union (synostosis) between the radius and ulna, the bony bridge is radically excised, and a vascularized adipofascial flap or synthetic barrier is interposed to prevent recurrence, often supplemented with a single dose of localized radiation or postoperative indomethacin.

Humeral and clavicular malunions are generally better tolerated due to the immense compensatory motion of the shoulder girdle, but severe cases require intervention. Symptomatic clavicular shortening (>2 cm) that alters scapular kinematics and causes thoracic outlet symptoms is treated via a superior approach. An opening wedge osteotomy is performed, a structural intercalary allograft is inserted to restore length, and robust superior plating is applied. Distal humeral malunions, such as the classic cubitus varus deformity following pediatric supracondylar fractures, are treated with a lateral closing wedge or dome osteotomy. The dome osteotomy is technically demanding but avoids the prominent lateral condylar bump associated with closing wedge techniques. Fixation is achieved with dual orthogonal or parallel locking plates to ensure sufficient stability for early elbow mobilization.

Pelvic Malunions

Pelvic malunions represent the zenith of orthopedic reconstructive complexity. These deformities result in severe mechanical back pain, sitting imbalance, and apparent limb length discrepancies due to hemipelvic migration. Correction often requires a massive, multidisciplinary three-stage reconstructive effort. Stage one involves an anterior approach (e.g., Pfannenstiel or ilioinguinal) to release contracted anterior structures and perform an osteotomy through the malunited symphysis pubis or pubic rami. Stage two requires repositioning the patient prone to access the posterior ring. The surgeon performs a posterior release, osteotomy of the malunited sacroiliac joint or sacral ala, and mobilizes the hemipelvis using heavy traction pins and specialized reduction clamps (e.g., Jungbluth clamps). Stage three involves definitive rigid fixation, typically utilizing lumbopelvic fixation, sacroiliac screws, and anterior symphyseal plating once the anatomical reduction of the pelvic ring is achieved. This is a highly morbid procedure reserved only for profoundly symptomatic patients.

Complications, Incidence Rates, and Salvage Management

The surgical correction of malunions is fraught with potential complications, driven by the compromised local biology, the presence of dense scar tissue, and the inherent biomechanical stresses placed on the fixation construct. The surgeon must be acutely aware of these risks and possess a comprehensive armamentarium of salvage strategies. Nonunion or delayed union at the osteotomy site is a primary concern, particularly in diaphyseal corrections where cortical contact may be minimal following angular or lengthening maneuvers. Infection is a catastrophic complication; superficial pin tract infections are ubiquitous with circular frames, but deep hardware infections following internal fixation require immediate aggressive intervention.

Neurovascular compromise is the most feared intra-operative complication. Acute correction of severe angular deformities or acute lengthening exceeding 2-3 centimeters places immense tension on tethered nerves and vessels. The common peroneal nerve is particularly vulnerable during valgus correction of the proximal tibia, and the median nerve is at risk during the mobilization of a severely shortened distal radius. Prophylactic nerve decompressions (e.g., fibular neck release for the peroneal nerve, carpal tunnel release for the median nerve) are frequently indicated. Hardware failure, including plate breakage or screw pull-out, occurs when the mechanical demands placed on the construct exceed its fatigue life before solid bony union is achieved, often due to patient non-compliance with weight-bearing restrictions.

Complication Estimated Incidence Salvage Management Strategy
Nonunion / Delayed Union 5% - 15% Optimization of biology (Vitamin D, smoking cessation). Revision surgery with decortication, robust autogenous bone grafting (RIA or iliac crest), and revision to a more stable fixation construct.
Pin Tract Infection (External Fixation) 20% - 40% Aggressive local pin care (chlorhexidine). Oral antibiotics for superficial erythema. Prompt pin removal and exchange for deep infections or loosening, combined with IV antibiotics.
Deep Hardware Infection (Internal Fixation) 2% - 5% Radical surgical debridement, hardware removal (if unstable), placement of antibiotic-impregnated cement spacers, culture-directed IV antibiotics, and eventual revision to external fixation.
Neurovascular Tethering / Palsy 1% - 3% Immediate cessation of acute correction. If post-operative, immediate release of dressings/splints. Surgical exploration and nerve decompression. Conversion to gradual correction via external fixator.
Hardware Failure / Loss of Fixation 3% - 8% Revision surgery. Utilization of longer plates, larger diameter intramedullary nails, or dual-plate constructs. Augmentation with structural allograft to share mechanical loads.
Complex Regional Pain Syndrome (CRPS) 2% - 10% (Upper Ext.) Multidisciplinary approach: aggressive hand therapy, neuropathic pain modulators (gabapentin), Vitamin C prophylaxis, and sympathetic nerve blocks (stellate ganglion block).

Phased Post-Operative Rehabilitation Protocols

The ultimate functional outcome of a meticulously executed malunion correction is inextricably linked to the rigor and appropriateness of the postoperative rehabilitation protocol. The rehabilitation strategy must be carefully phased, balancing the competing demands of protecting the osteotomy and fixation construct with the necessity of early mobilization to prevent debilitating joint stiffness and soft-tissue contractures.

Phase 1: Immediate Post-Operative Phase (Weeks 0-2)
The primary goals during this phase are the protection of the surgical site, meticulous wound care, and the management of edema and pain. For internal fixation constructs, the limb is typically immobilized in a well-padded splint. Elevation is critical. Active and passive range of motion (ROM) of all joints not spanned by the fixation is initiated immediately. For example, following a distal radius osteotomy, aggressive digital ROM, elbow flexion/extension, and shoulder circumduction are mandatory to prevent complex regional pain syndrome (CRPS) and tendon adhesions. Weight-bearing is strictly prohibited for lower extremity osteotomies during this phase.

Phase 2: Callus Formation and Early Mobilization Phase (Weeks 2-6)
Once the surgical incisions have healed and sutures are removed, the rehabilitation focus shifts to restoring the ROM of the adjacent joints. If rigid internal fixation (e.g., locking plates or statically locked IM nails) was achieved, the splint is transitioned to a removable brace, and formal physical therapy commences. Gentle active-assisted ROM is initiated. For lower extremity malunions, touch-down weight-bearing (TDWB) or partial weight-bearing (PWB) may be permitted depending on the inherent stability of the fracture pattern and the robustness of the hardware. For patients undergoing distraction osteogenesis via circular frames, this phase coincides with the active distraction phase (typically 1 mm per day). Intensive physical therapy is required to stretch the muscles crossing the lengthening segment to prevent severe joint contractures (e.g., equinus contracture of the ankle during tibial lengthening).

Phase 3: Consolidation and Strengthening Phase (Weeks 6-12)
This phase is guided by radiographic evidence of healing. Once bridging callus is clearly visible on orthogonal radiographs across at least three of the four cortices, the rehabilitation protocol is aggressively advanced. Weight-bearing is progressively increased to full weight-bearing as tolerated (FWBAT). The physical therapy focus shifts from pure ROM to progressive resistance exercises, core stabilization, and gait retraining. Closed kinetic chain exercises are prioritized to stimulate mechanoreceptors and promote further bone remodeling according to Wolff's Law.

Phase 4: Return to Function and Frame Removal (Weeks 12+)
For internal fixation, patients continue strengthening and transition back to their baseline occupational and recreational activities. For patients with circular external fixators, the frame remains in place until the regenerate bone has fully consolidated and corticalized. Prior to frame removal, a "dynamization" protocol is often employed, where the frame is mechanically loosened to allow axial loading of the regenerate bone, stimulating hypertrophy. Once the frame is removed, the limb is temporarily protected in a functional brace or cast for 4-6 weeks to prevent late deformation or fracture of the newly formed bone.

Summary of Landmark Literature and Clinical Guidelines

The modern surgical management of malunited fractures is built upon a foundation of landmark biomechanical studies and clinical trials. Dror Paley’s seminal text, Principles of Deformity Correction, remains the definitive orthopedic bible for understanding the


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