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Ankle Fractures: Comprehensive Guide to Epidemiology, Classification, Anatomy & Biomechanics

Operative Management of Deltoid Ligament Tears and Lateral Malleolar Fractures

01 May 2026 20 min read 41 Views
Operative Management of Deltoid Ligament Tears and Lateral Malleolar Fractures

Key Takeaway

A deltoid ligament tear combined with a lateral malleolar fracture represents an unstable ankle injury equivalent to a bimalleolar fracture. Driven by a supination-external rotation mechanism, this injury compromises the tibiotalar contact area. Accurate diagnosis relies on stress radiography rather than clinical medial tenderness. Surgical management primarily involves anatomic open reduction and internal fixation of the fibula, with medial exploration reserved for cases of blocked reduction or persistent mortise widening.

Comprehensive Introduction and Patho-Epidemiology

A deltoid ligament tear accompanied by a fracture of the lateral malleolus represents a highly unstable ankle injury, functionally and radiographically equivalent to a bimalleolar fracture. This specific injury pattern is predominantly driven by the exact same biomechanical mechanism that produces classic bimalleolar fractures: supination of the foot coupled with an external rotation force, classically categorized as a Lauge-Hansen Supination-External Rotation (SER) Stage IV injury. However, in this bimalleolar-equivalent variant, instead of the medial malleolus failing under tensile forces to produce a bony avulsion, the deltoid ligament complex ruptures. This ligamentous failure allows the talus to displace laterally and rotate externally within the mortise, effectively destroying the medial restraint of the ankle joint. Concomitantly, the anterior capsule of the ankle joint is frequently torn, further exacerbating the multi-planar instability of the tibiotalar articulation.

The epidemiology of ankle fractures dictates that they are among the most common injuries treated by orthopedic surgeons, with an incidence of approximately 187 per 100,000 persons annually. Within this massive cohort, the Lauge-Hansen SER mechanism accounts for roughly 40% to 70% of all ankle fractures. The subset of SER fractures that manifest as isolated lateral malleolus fractures with a concurrent deltoid ligament rupture (SER IV bimalleolar equivalents) represents a significant diagnostic and therapeutic challenge. These injuries exhibit a bimodal distribution, frequently affecting young, active males secondary to high-energy sports trauma, and older, osteoporotic females following low-energy twisting falls. The recognition of the medial-sided soft tissue injury is paramount; failure to identify the deltoid rupture leads to the misclassification of an unstable SER IV injury as a stable SER II injury, a catastrophic diagnostic error.

The pathogenesis of this injury follows a predictable, sequential failure of the osseous and ligamentous restraints of the ankle. As the supinated foot is subjected to an external rotation force, the anterior inferior tibiofibular ligament (AITFL) is the first structure to fail (SER Stage I). As the force continues, a short oblique or spiral fracture propagates through the lateral malleolus, typically originating at the level of the tibial plafond and extending posterosuperiorly (SER Stage II). Further external rotation leads to the rupture of the posterior inferior tibiofibular ligament (PITFL) or an avulsion fracture of the posterior malleolus (SER Stage III). Finally, the terminal event in this sequence is the failure of the medial structures. The talus forcefully externally rotates and abducts against the medial malleolus, causing either a transverse avulsion fracture of the medial malleolus or a complete rupture of the deltoid ligament complex (SER Stage IV).

The clinical significance of this bimalleolar-equivalent injury cannot be overstated. Historically, the "hidden" nature of the medial ligamentous injury led to high rates of non-operative management for what appeared to be isolated, stable fibular fractures. As the initial hematoma and swelling subsided, the unconstrained talus would inevitably subluxate laterally within the cast. This occult displacement drastically alters the contact mechanics of the tibiotalar joint. The modern orthopedic consensus recognizes that the bimalleolar-equivalent fracture is a highly unstable entity that demands meticulous evaluation, dynamic stress testing, and, in the vast majority of cases, operative stabilization to prevent the rapid onset of post-traumatic osteoarthritis.

Detailed Surgical Anatomy and Biomechanics

Osteology and the Ankle Mortise

The ankle joint is a highly congruent, modified hinge joint formed by the articulation of the talus with the distal tibia and fibula. The mortise is uniquely trapezoidal, being wider anteriorly than posteriorly. This anatomical nuance dictates that the ankle is most stable in maximal dorsiflexion, where the widest portion of the talar dome is wedged into the mortise, tensioning the syndesmotic ligaments. The lateral malleolus extends further distally and posteriorly than the medial malleolus, acting as a crucial lateral buttress that prevents lateral talar translation and external rotation. The fibula also serves as the origin for the lateral collateral ligaments and the lateral half of the syndesmotic complex.

Anatomy of the Deltoid Ligament Complex

The deltoid ligament is a robust, multifascicular, fan-shaped structure that serves as the primary medial stabilizer of the ankle. It is anatomically and functionally divided into superficial and deep components. The superficial deltoid originates from the anterior colliculus of the medial malleolus and spans across two joints. It comprises the tibionavicular, tibiocalcaneal, and superficial posterior tibiotalar ligaments. The superficial fibers primarily resist hindfoot eversion and provide a secondary restraint to external rotation. Because they cross the subtalar joint, they also play a role in maintaining the medial longitudinal arch of the foot.

The deep deltoid ligament is intra-articular but extrasynovial, originating from the posterior colliculus and the intercollicular groove of the medial malleolus. It comprises the anterior and deep posterior tibiotalar ligaments. The deep posterior tibiotalar ligament is a short, exceedingly thick, and horizontally oriented band. It is universally recognized as the most critical component of the medial ankle complex. The deep deltoid is the primary restraint against lateral displacement and external rotation of the talus. A complete rupture of the deep deltoid ligament uncouples the talus from the medial malleolus, rendering the ankle mortise fundamentally unstable regardless of the integrity of the superficial fibers.

Biomechanical Consequences of Displacement

The tibiotalar joint relies on an exquisite degree of articular congruency to distribute weight-bearing loads, which can exceed five times body weight during normal ambulation. Even microscopic deviations in talar positioning drastically alter joint contact mechanics. Landmark biomechanical studies by Ramsey and Hamilton demonstrated that a mere 1-mm lateral shift of the talus reduces the effective weight-bearing contact area of the tibiotalar articulation by 42%. A 5-mm lateral shift reduces the contact area by up to 80%.

This exponential decrease in contact area leads to a proportional, massive increase in peak articular cartilage stress. The cartilage of the ankle joint is relatively thin compared to the knee or hip, making it highly susceptible to mechanical overload. When the talus shifts laterally due to an unaddressed deltoid rupture and fibular shortening, the load is concentrated on the lateral shoulder of the talar dome and the medial articular facet of the fibula. This abnormal load distribution rapidly outstrips the reparative capacity of the chondrocytes, predisposing the patient to early, severe post-traumatic osteoarthritis if anatomic reduction is not achieved and rigorously maintained.

Exhaustive Indications and Contraindications

The management of SER-type lateral malleolar fractures with concurrent deltoid ligament tears requires meticulous patient selection and a thorough understanding of fracture stability. The primary goal of treatment is the absolute restoration of the ankle mortise congruity to prevent degenerative joint disease. While operative intervention is the gold standard for active patients, various systemic and local factors must be weighed.

Operative Indications

The absolute indication for operative intervention (Open Reduction and Internal Fixation [ORIF] of the fibula) is a demonstrably unstable ankle mortise. This is defined radiographically as a medial clear space measuring greater than 4 mm on a dynamic stress radiograph (gravity or manual stress), or asymmetric widening of the medial clear space compared to the superior clear space. Furthermore, any frank lateral or posterior subluxation of the talus on static resting radiographs mandates surgical stabilization. Open fractures, fractures associated with vascular compromise, and fractures presenting with impending skin necrosis (tenting) represent surgical emergencies requiring immediate operative management.

Relative Indications and the Non-Operative Debate

Relative indications for surgery include high-demand athletes or manual laborers who require an accelerated return to function, even if the stress radiograph is only marginally positive. Conversely, non-operative management may be considered in a highly specific subset of patients. Recent literature has identified the "stress-positive only" fracture—where the mortise is perfectly congruent on resting static views but widens only under stress. In highly compliant, lower-demand patients, closed treatment with a well-molded short-leg cast and strict non-weight-bearing can yield outcomes equivalent to ORIF, provided serial radiographs confirm absolutely no displacement over a 6-week period. However, the risk of late displacement remains a significant clinical anxiety, making surgery the preferred route for most surgeons.

Contraindications to Surgical Intervention

Absolute contraindications to immediate internal fixation include active deep infection at the surgical site and a prohibitive soft-tissue envelope, such as the presence of massive fracture blisters, severe edema preventing wound closure, or active cellulitis. In these scenarios, the ankle should be spanned with a temporary external fixator until the soft tissues declare themselves. Severe peripheral arterial disease (e.g., absent pedal pulses, ABI < 0.5) is a strong relative contraindication, as surgical incisions may fail to heal, leading to catastrophic limb loss. Patients with acute Charcot neuroarthropathy require specialized, highly rigid constructs (often fine-wire circular frames or tibiotalocalcaneal nailing) rather than standard plate-and-screw constructs, which will inevitably fail in the neuropathic patient.

Category Specific Condition Rationale / Management Strategy
Absolute Indications Medial clear space > 4mm on stress view Confirms deep deltoid rupture and mortise instability; requires lateral buttress restoration.
Absolute Indications Talar subluxation on static resting X-ray Indicates gross instability; closed reduction will not be maintained as swelling subsides.
Relative Indications High-demand athlete / Manual laborer Accelerated rehabilitation; minimizes risk of late displacement and prolonged immobilization.
Contraindications Severe soft-tissue compromise (blisters) High risk of wound dehiscence and deep infection. Delay definitive ORIF or use external fixation.
Contraindications Severe Peripheral Arterial Disease Incisions over the distal fibula have tenuous blood supply; high risk of ischemic necrosis.
Contraindications Acute Charcot Neuroarthropathy Standard hardware will fail due to lack of protective sensation and osteoclastic overactivity.

Pre-Operative Planning, Templating, and Patient Positioning

Diagnostic Imaging and Stress Radiography

Pre-operative planning begins with high-quality, orthogonal radiographs of the injured ankle, including anteroposterior (AP), lateral, and mortise views. The mortise view, obtained with the leg internally rotated 15 to 20 degrees, allows for the clear visualization of the entire articular space. If the static radiographs demonstrate an isolated lateral malleolus fracture with a congruent mortise, dynamic stress imaging is mandatory to unmask an occult deltoid ligament tear.

Gravity external rotation stress radiography is the preferred modality. The patient is placed in the lateral decubitus position with the affected leg elevated and the ankle unsupported, allowing gravity to externally rotate the foot. This technique is highly reproducible, less painful for the patient than manual stress, and eliminates radiation exposure to the physician. It is critical that the ankle is held in neutral dorsiflexion during stress views; plantar flexion places the narrowest posterior portion of the talar dome within the mortise, artificially widening the medial clear space and yielding a false-positive result. Advanced imaging, such as MRI, is exquisitely sensitive for deltoid tears but is rarely indicated in the acute fracture setting, as dynamic radiography provides the functional assessment required for surgical decision-making.

Surgical Templating and Soft Tissue Assessment

Pre-operative templating ensures appropriate implant availability and reduces intraoperative delays. The surgeon must evaluate the fracture morphology (Danis-Weber classification) to determine the optimal fixation construct. For standard oblique SER fractures, templating typically involves a 3.5-mm cortical lag screw for interfragmentary compression, protected by a one-third tubular neutralization plate. For osteoporotic bone or highly comminuted fractures, pre-contoured anatomic locking plates should be templated.

The soft tissue envelope dictates the timing of surgery. The "wrinkle test" is the gold standard clinical assessment: if the skin over the lateral malleolus wrinkles when the ankle is dorsiflexed or everted, the edema has sufficiently subsided to allow for safe surgical incision and closure. Operating through massive edema or hemorrhagic fracture blisters drastically increases the risk of catastrophic wound dehiscence.

Patient Positioning and Operating Room Setup

The patient is positioned supine on a fully radiolucent operating table. A sandbag or gel bump is placed under the ipsilateral hip to internally rotate the operative leg, bringing the lateral malleolus parallel to the floor and perfectly profiling the fibula for the surgeon. A well-padded thigh or calf tourniquet is applied to ensure a bloodless surgical field, optimizing visualization of small articular fragments and interposed soft tissues.

The fluoroscopy unit (C-arm) is positioned on the contralateral side of the table. The monitor should be placed in the direct line of sight of the primary surgeon. The setup must allow for unhindered, rapid acquisition of AP, mortise, and lateral images without compromising the sterile field. Pre-draping prophylactic intravenous antibiotics (typically a first-generation cephalosporin) must be administered within one hour prior to tourniquet inflation.

Step-by-Step Surgical Approach and Fixation Technique

Approach to the Lateral Malleolus

The operative sequence begins with the anatomic restoration of the lateral malleolus, which indirectly reduces the talus and restores the lateral buttress of the ankle mortise. A longitudinal or slightly posterolateral incision is made over the distal fibula, centered precisely over the fracture site. The incision extends through the epidermis and dermis. Subcutaneous tissues are sharply divided in line with the incision.

Meticulous soft tissue handling is critical. The surgeon must identify and protect the superficial peroneal nerve anteriorly and the sural nerve posteriorly. The periosteum is incised longitudinally and elevated minimally—only 1 to 2 millimeters from the fracture edges. Over-stripping the periosteum devitalizes the cortical bone fragments, significantly delaying union and increasing the risk of hardware failure. The fracture hematoma is thoroughly evacuated using a curette and irrigation. The fracture ends are meticulously debrided of any interposed soft tissue, periosteum, or loose osteochondral fragments.

Fixation of the Lateral Malleolus

Anatomic reduction of the fibula is the cornerstone of the procedure. The fracture is reduced using pointed reduction forceps, ensuring absolute restoration of fibular length, alignment, and rotation. For the classic long oblique or spiral SER fracture, fixation is achieved utilizing the lag screw principle. A 3.5-mm gliding hole is drilled in the near cortex, followed by a 2.5-mm thread hole in the far cortex. A 3.5-mm cortical screw is placed perpendicular to the fracture plane to generate maximal interfragmentary compression.

Once compressed, the fracture is protected with a neutralization plate. A one-third tubular plate or a pre-contoured anatomic locking plate is applied to the lateral or posterolateral surface of the fibula to neutralize torsional, bending, and shear forces that would otherwise cause the lag screw to fail. The plate is secured with 3.5-mm cortical screws proximally in the diaphyseal bone and 3.5-mm or 4.0-mm cancellous or locking screws in the softer metaphyseal bone distally. For short oblique fractures, a posterolateral antiglide plate is biomechanically superior. This construct acts as a buttress against proximal migration of the distal fragment and allows screws to be placed from posterior to anterior, capturing the thicker bone of the distal fibula and avoiding intra-articular penetration.

Evaluation of the Mortise and Medial Exploration

Following rigid fixation of the fibula, intraoperative fluoroscopy (mortise view) is utilized to assess the medial clear space. In the vast majority of cases, restoring the lateral buttress pulls the talus laterally, anatomically reducing the medial clear space to less than 4 mm.

If the medial clear space remains persistently widened (>4 mm) or is asymmetric compared to the superior clear space despite anatomic fibular fixation, soft tissue interposition is highly likely. This is an absolute indication for medial exploration. A 4- to 5-cm longitudinal incision is made centered over the medial malleolus. The saphenous vein and nerve are identified and retracted anteriorly. The ankle joint capsule is opened. The surgeon will typically find the deep deltoid ligament avulsed from its tibial attachment and flipped into the joint space, blocking reduction. The entrapped ligament is extracted from the mortise using a Freer elevator or skin hook. Once the mortise is cleared, the talus should reduce anatomically. While routine repair of the deltoid is not strictly necessary once the mortise is reduced, many surgeons opt to repair the avulsed ligament using suture anchors placed into the medial malleolus, securing the deep and superficial fibers with heavy non-absorbable sutures to augment medial stability.

Syndesmotic Evaluation

The final intraoperative step is the evaluation of the distal tibiofibular syndesmosis. The Lauge-Hansen SER mechanism inherently involves tearing of the AITFL, and the syndesmosis may remain unstable even after fibular fixation. A "Cotton test" (lateral traction applied to the fibula with a bone hook) or an external rotation stress test is performed under live fluoroscopy. If the tibiofibular clear space widens, the syndesmosis is unstable. It must be anatomically reduced using a large pelvic reduction clamp and stabilized with either trans-syndesmotic screws (typically two 3.5-mm or 4.5-mm screws across three or four cortices) or a dynamic suture-button construct.

Complications, Incidence Rates, and Salvage Management

Despite meticulous surgical technique, the operative management of bimalleolar-equivalent fractures carries a distinct profile of complications. The surgeon must be prepared to identify and manage these adverse events to optimize long-term patient outcomes.

Malreduction and Talar Shift

The most catastrophic technical complication is the malreduction of the lateral malleolus. Failure to restore the exact length and rotation of the fibula results in a persistent lateral talar shift. Even a 1-mm residual subluxation alters joint kinematics profoundly. Incidence rates of malreduction in literature range from 2% to 5% in high-volume centers. If identified early post-operatively, immediate revision ORIF is mandatory. If identified late (after fracture consolidation), salvage management requires a complex fibular lengthening osteotomy and potential medial soft tissue reconstruction. If severe post-traumatic arthritis has already developed, salvage is limited to ankle arthrodesis or total ankle arthroplasty.

Wound Complications and Infection

The lateral fibula has a tenuous blood supply and minimal soft tissue coverage, making it highly susceptible to wound healing issues. Superficial wound dehiscence occurs in approximately 5% to 10% of cases, while deep surgical site infections occur in 1% to 2%. Patient-specific risk factors, notably poorly controlled diabetes mellitus and active tobacco use, exponentially increase these rates. Superficial necrosis can often be managed with local wound care and oral antibiotics. Deep infections require aggressive surgical debridement, hardware removal (if the fracture is united) or conversion to external fixation (if non-united), and prolonged culture-directed intravenous antibiotic therapy.

Post-Traumatic Osteoarthritis

Even with perfect anatomic reduction and uncomplicated healing, the initial impact of the trauma causes irreversible damage to the chondrocytes of the talar dome and tibial plafond. The incidence of radiographic post-traumatic osteoarthritis following operatively treated SER fractures is reported to be between 10% and 40% at 10-year follow-up, though symptomatic arthritis requiring intervention is significantly lower. The risk is directly correlated with the initial energy of the injury and the accuracy of the surgical reduction.

Complication Estimated Incidence Primary Prevention Salvage Management Strategy
Fibular Malreduction 2% - 5% Meticulous intra-op fluoroscopy; anatomic lag screw technique. Early: Revision ORIF. Late: Fibular lengthening osteotomy.
Superficial Dehiscence 5% - 10% Delay surgery until "wrinkle test" is positive; gentle soft tissue handling. Local wound care; negative pressure wound therapy; oral antibiotics.
Deep Infection 1% - 2% Pre-op prophylactic antibiotics; optimized glycemic control. Serial I&D; hardware removal; IV antibiotics; potential flap coverage.
Symptomatic Hardware 15% - 30% Use of low-profile plates; countersinking screw heads. Hardware removal after complete fracture consolidation (minimum 6-12 months).
Post-Traumatic Arthritis 10% - 40% (Radiographic) Absolute anatomic restoration of the mortise; early ROM. Activity modification; bracing; Ankle Arthrodesis; Total Ankle Arthroplasty.

Phased Post-Operative Rehabilitation Protocols

The post-operative rehabilitation protocol following ORIF of a bimalleolar-equivalent fracture must strike a delicate balance between protecting the surgical fixation and preventing debilitating joint stiffness. The protocol is generally divided into distinct phases based on the biology of bone and ligament healing.

Phase I: Maximum Protection (0 to 2 Weeks)

Immediately following surgery, the patient is placed in a well-padded, bulky short-leg splint (e.g., a posterior slab with a U-splint) with the ankle held in strict neutral dorsiflexion. Equinus positioning must be avoided to prevent Achilles tendon contracture. The primary goals of this phase are wound healing, edema control, and pain management. The patient remains strictly non-weight-bearing (NWB) on the operative extremity. Elevation of the limb above the level of the heart is emphasized to mitigate swelling, which directly reduces tension on the surgical incision.

Phase II: Early Motion and Controlled Loading (2 to 6 Weeks)

At the two-week post-operative mark, the patient returns to the clinic for suture or staple removal, provided the wound is completely healed. The patient is then transitioned from the rigid splint to a removable controlled ankle motion (CAM) boot. The critical transition in this phase is the initiation of active range of motion (ROM) exercises. The patient is instructed to remove the boot multiple times daily to perform active dorsiflexion, plantar flexion, inversion, and eversion. Early motion is vital to nourish the articular cartilage via synovial fluid diffusion and to prevent capsular contracture. Weight-bearing status in this phase is highly dependent on bone quality, fracture comminution, and the rigidity of the fixation. Young patients with robust bone may begin progressive weight-bearing in the CAM boot, while osteoporotic or non-compliant patients typically remain NWB or touch-down weight-bearing (TDWB) until the 6-week mark.

Phase III: Progressive Strengthening (6 to 12 Weeks)

At 6 weeks post-operatively, orthogonal radiographs are obtained to confirm clinical and radiographic union of the fibula. Once bridging callus is visualized and the fracture is non-tender to palpation, the patient is progressively weaned from the CAM boot into a supportive, lace-up athletic shoe. Formal physical therapy is aggressively pursued during this phase. Rehabilitation focuses on restoring full, symmetric ankle ROM, proprioceptive retraining (e.g., balance board exercises) to compensate for the disrupted ligamentous mechanoreceptors, and strengthening of the peroneal musculature to provide dynamic lateral ankle stability.

Phase IV: Return to High-Level Function (3 to 6+ Months)

The final phase of rehabilitation focuses on returning the patient to their pre-injury level of occupational and athletic activity. Patients progress to plyometric exercises, sport-specific cutting drills, and endurance training. While the bone is fully united, patients must be counseled that maximal medical improvement, particularly regarding residual swelling and soft-tissue induration, may take up to 12 to 18 months. Continued use of a lace-up ankle brace during high-impact sports is often recommended for the first year post-injury.

Summary of Landmark Literature and Clinical Guidelines

The operative management of deltoid ligament tears and lateral malleolar fractures is heavily guided by a robust body of orthopedic literature. Understanding these landmark studies is essential for evidence-based surgical practice.

Biomechanical Foundations

The absolute necessity of anatomic reduction was definitively established by Ramsey and Hamilton in their 1976 landmark paper published in the Journal of Bone and Joint Surgery. Their cadaveric study mathematically quantified the relationship between lateral talar shift and tibiotalar contact area, famously demonstrating that a 1-mm lateral shift reduces contact area by 42%. This single data point remains the most frequently cited biomechanical justification for the operative intervention of unstable ankle fractures, shifting the paradigm away from closed casting for displaced injuries.

The Role of Stress Radiography

The diagnostic algorithm for isolated lateral malleolar fractures was revolutionized by the work of Michelson et al., who elucidated the unreliability of clinical examination in diagnosing deltoid ligament ruptures. Historically, medial ecchymosis and tenderness were considered pathognomonic for a deltoid tear. However, modern prospective studies have definitively proven that clinical medial tenderness has a positive predictive value of less than 60% for actual deep deltoid disruption. This literature mandated the universal adoption of dynamic stress radiography (gravity or manual) as the gold standard for differentiating stable SER II from unstable SER IV injuries.

Controversies in Deltoid Repair

The debate regarding the necessity of primary deltoid ligament repair during fibular ORIF has been addressed by multiple randomized controlled trials. Historically, some surgeons advocated for routine medial exploration and suture repair of the deltoid. However, landmark studies, including those by Stromsoe et al. and more recent meta-analyses, have demonstrated no statistically significant difference in long-term functional outcomes, pain scores, or radiographic stability between patients who underwent fibular ORIF alone versus those who underwent fibular ORIF with concurrent deltoid repair. Consequently, current clinical guidelines dictate that the deltoid ligament does not require routine repair, provided the mortise reduces anatomically following fibular fixation. Medial exploration is strictly reserved for cases of mechanical block to reduction.

Non-Operative Management of Stress-Positive Fractures

Recent literature has introduced nuance to the absolute indication for surgery in stress-positive fractures. Studies by Sanders et al. and the HSS orthopaedic group have investigated the non-operative management of fractures that are perfectly aligned on static views but widen on stress views. Their data suggest that if anatomic alignment is strictly maintained in a cast, one-year functional outcomes can be equivalent to ORIF. However, these studies emphasize that this approach requires exhaustive patient compliance, weekly radiographic surveillance, and a low threshold for surgical conversion if even 1 mm of displacement occurs. For the general orthopedic practitioner, operative stabilization remains the most reliable method to guarantee mortise congruity and prevent long-term morbidity.

Clinic OS
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Prof. Clinic OS
Consultant Orthopedic & Spine Surgeon
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